Evolution and Analysis
Originating from Switzerland, the Biocell Ultravital brand stands out for its profound contributions to healthcare by developing cutting-edge formulas that optimize cellular functions based on decades of extensive research. Our therapies provide a wide range of benefits, including anti-aging process, cell revitalization, inflammation control, hormonal balance, detoxification, and metabolic regulation. Globally, for over 70 years, our therapies have improved the cellular health of thousands of patients and advanced the use of preventive and regenerative medicine in clinical practice. At Biocell Ultraviral, we seek to establish a legacy of “Lengthening Life with Health.”
Biotechnology is evolving rapidly from its traditional focus on research and development to encompass manufacturing, sales, and marketing. Key growth areas in biotechnology include DNA chips, nanotechnology, proteomics, and regenerative medicine, such as stem cell therapy and tissue engineering.
Because it generally focuses on symptoms, pharmacological therapy is oriented towards the final links of the disease, so thus, it is more symptomatic than causal, and it is pathogenetically and therapeutically directed towards the consequences and not towards rectifying whether the failure is due to the mitochondrial, ribosomal or cyto-membranes functions being misdirected or genetically programmed causing cellular disorders.
A major drawback of conventional pharmacology therapy is that it often targets the symptoms rather than the underlying causes of a disease. Addressing the later stages of the disease (i.e., symptomatic phase) neglects the fundamental cellular dysfunction in the mitochondria, ribosome, or cytomembrane that occurs due to aging or genetic defects.
Throughout life, body tissues naturally endure wear and tear, but they possess an intrinsic ability to self-renew and repair worn structures. Without this self-renewal capability, the lifespan of organisms would diminish drastically. Every living organism is comprised of cells carrying the same genetic code, although their behavior may differ. For instance, the cell cycle controls growth and replication, but it is governed by various restriction points that may halt the cell cycle in the event of nutrient scarcity, DNA damage, or external chemical damage. The balance between cell growth and programmed death (i.e., apoptosis) is complex and involves numerous genes.
The p53 gene, in particular, plays a crucial role in stopping the cell cycle when DNA damage is detected, allowing time for repair. If the damage is irreparable, p53 initiates apoptotic death of the damaged cell before it accumulates DNA mutations. Essentially, p53 protects against the proliferation of potentially cancerous cells, maintaining cellular and genetic stability. Hence, defective p53 can lead to genomic instability, more frequent mutations, and an increased risk of cancer development. In fact, this gene is found to be mutated in over 50% of all human cancers.
The previous example illustrates how cellular disorders can lead to difficult-to-treat diseases. While many factors can trigger these disorders, they primarily stem from cell dysfunction and deterioration of the cell microenvironment. Indeed, most human diseases originate from cell degeneration and subsequent tissue death, manifesting either suddenly, as in heart attacks, or gradually through chronic diseases and aging.